Tricuspid regurgitation

Overview tricuspid regurgitation may be relative and two organic. Relative, the valve itself has no lesions, and the Department of right ventricular hypertrophy, atrioventricular Central corresponding expansion tricuspid valve caused a bad right, causing regurgitation. Severe rheumatic mitral stenosis or regurgitation in patients accompanied relative tricuspid regurgitation. Organic tricuspid regurgitation of rheumatic fever in the aftermath of clinical rare, Most accompanied by mitral and aortic valve disease. Pathological changes of fiber valve thickening, kink, tendons shorten expansion valve, cardiac valve when a not completely right. Often combined valve and the junction of integration, which both narrow. Cause pathogenesis tricuspid regurgitation is the pathophysiology of tricuspid regurgitation, that is, from right ventricular systolic flow back into the right atrium and right atrium caused high expansion, the increase of pressure, venous return obstacles. As of right ventricular overload, compensatory and hypertrophy, right heart failure prone. Clinical manifestations of the tricuspid valve insufficiency with the signs and symptoms of mitral insufficiency related to the degree. Mild regurgitation clinically detectable difficult. In more severe cases can be tired and poor Be selective, liver pain, abdominal swelling and lower extremity edema. Typical signs : jugular venous engorgement with pulses; Hepatomegaly and can palpable pulse; and the left margin sternum 4 intercostal the entire briefing-systolic murmur. inspiratory noise in the deep end of enhancement (Bahrain levy). Typical signs of severe tricuspid regurgitation in patients can be absent. If long-term liver blood where Yu sclerosis, but no longer throb; Right ventricular volume load achieve pole, With inspiratory noise no longer enhanced, it can levy Bahrain negative. X-ray photographs show the right atrium and right ventricular hypertrophy and heart right edge abdomen, while the other half are caused by the valve lesions change. ECG said atrial hypertrophy, P wave height and width; And the right bundle branch block or right ventricular hypertrophy, or myocardial injury. Often atrial fibrillation. Echocardiography and Doppler examination : a plane ultrasound can detect the size of the tricuspid annulus and understand the thickening of the valve. help at first identified relativity and organic diseases. Tricuspid valve insufficiency, ultrasound contrast microbubbles can be seen from the tricuspid; Doppler can monitor directly to the right ventricle to the right atrium of abnormal signal and can estimate the extent of reflux. Cardiac catheterization showed right atrial pressure waveform in a V-prominent, y descending steep, and the inspiration is more obvious. Right atrial pressure waveform and right ventricular pressure waveform similar, only smaller amplitude, known as the right ventricle of the right atrial pressure, is a severe tricuspid regurgitation performance. Cardiovascular imaging : the right ventricular angiography, right anterior oblique film photography can show that the extent and tricuspid regurgitation. However, cardiac catheterization across the tricuspid valve, a potential false positive. Tricuspid valve insufficiency diagnosis should include the right degree of incompetence understanding. Typical clinical signs in the diagnosis of severe tricuspid regurgitation have a certain value. Past with right ventricular angiography as a diagnostic suspicious cases and the estimated regurgitation means. In recent years, ultrasound and Doppler examination has been gradually replaced by the traumatic examination.