Children with acute hemiplegia refers to acute hemiplegia acquired, by a variety of causes of a syndrome. The main clinical manifestations of acute hemiplegia, often accompanied by seizures, aphasia, mental retardation or behavioral abnormalities.
Etiology and Pathogenesis:
Chinese medicine believes that the disease is mainly external exogenous evil, blocking the meridians, internal, compared with less than innate endowment, acquired nursed lost in. Modern medicine that this disease usually caused by occlusive cerebrovascular disease caused by local cerebral ischemia or necrosis due to cerebral artery disease is most common. Cause of the infection most vascular brain inflammation caused by allergic reactions caused by direct invasion or infection by pathogenic microorganisms after. Mycobacterium tuberculosis, pyogenic bacteria, viruses, Leptospira infection can cause paralysis, caused by Mycobacterium tuberculosis which are more common. After infection or after vaccination can cause perivascular inflammation due to allergy caused paralysis. Seizures in children with brain injury is another common cause of acute hemiplegia, which status epilepticus caused by the most common. Cerebral vascular malformations, brain trauma, nerve skin syndrome, homocystinuria, brain tumors can occur hemiplegia, or paralysis as the main performance.
Clinical manifestations:
There are three children with acute onset hemiplegia ways:
① stroke type, the most common, sudden convulsions, high fever, coma, hemiplegia, and sometimes convulsions was status epilepticus. Side visual impairment, language barriers exist with hemiplegia.
② acute type, showed only hemiplegia, no change or a mere consciousness of consciousness.
③ interval type, transient limb weakness, common carotid artery occlusion, contralateral paralysis if the carotid pulse weakened, with diagnostic significance. Onset cases determines the nature of the lesion, blood clots may onset within minutes; thrombosis onset is slow, intermittent or in a few hours or a few days aggravated sexual, but most no convulsions; hemangioma rupture caused by rapid onset, sudden head pain, pale, cold sweats.
Paralysis often occur after convulsions stopped. About 3 to 4 months motor function gradually recovered, lower limb functional recovery faster than the upper limbs, about half left with varying degrees of sequelae, severe limb deformities. If the basal ganglia of the brain damage occurs involuntary movement of the hand, foot or dance-like movements; facial paralysis occurs when cranial nerve damage, difficulty swallowing, drooling and pronunciation speech is unclear; if the temporal lobe, occipital lobe or the parietal lobe damage can occur in vision disorders, aphasia or sensory disorders; more severe cases, may have intelligence and behavioral disorders.
Etiology and Pathogenesis:
Chinese medicine believes that the disease is mainly external exogenous evil, blocking the meridians, internal, compared with less than innate endowment, acquired nursed lost in. Modern medicine that this disease usually caused by occlusive cerebrovascular disease caused by local cerebral ischemia or necrosis due to cerebral artery disease is most common. Cause of the infection most vascular brain inflammation caused by allergic reactions caused by direct invasion or infection by pathogenic microorganisms after. Mycobacterium tuberculosis, pyogenic bacteria, viruses, Leptospira infection can cause paralysis, caused by Mycobacterium tuberculosis which are more common. After infection or after vaccination can cause perivascular inflammation due to allergy caused paralysis. Seizures in children with brain injury is another common cause of acute hemiplegia, which status epilepticus caused by the most common. Cerebral vascular malformations, brain trauma, nerve skin syndrome, homocystinuria, brain tumors can occur hemiplegia, or paralysis as the main performance.
Clinical manifestations:
There are three children with acute onset hemiplegia ways:
① stroke type, the most common, sudden convulsions, high fever, coma, hemiplegia, and sometimes convulsions was status epilepticus. Side visual impairment, language barriers exist with hemiplegia.
② acute type, showed only hemiplegia, no change or a mere consciousness of consciousness.
③ interval type, transient limb weakness, common carotid artery occlusion, contralateral paralysis if the carotid pulse weakened, with diagnostic significance. Onset cases determines the nature of the lesion, blood clots may onset within minutes; thrombosis onset is slow, intermittent or in a few hours or a few days aggravated sexual, but most no convulsions; hemangioma rupture caused by rapid onset, sudden head pain, pale, cold sweats.
Paralysis often occur after convulsions stopped. About 3 to 4 months motor function gradually recovered, lower limb functional recovery faster than the upper limbs, about half left with varying degrees of sequelae, severe limb deformities. If the basal ganglia of the brain damage occurs involuntary movement of the hand, foot or dance-like movements; facial paralysis occurs when cranial nerve damage, difficulty swallowing, drooling and pronunciation speech is unclear; if the temporal lobe, occipital lobe or the parietal lobe damage can occur in vision disorders, aphasia or sensory disorders; more severe cases, may have intelligence and behavioral disorders.
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